Abstract
Environmental noise (originating mainly from traffic) has been implicated as a risk factor for adverse cardiovascular outcomes. Systematic reviews of the associations between noise from transportation-related sources (e.g., road, air and rail) and cardiovascular diseases concluded that individuals exposed to higher levels of noise are at increased risk for hypertension and ischemic heart disease, making exposure to traffic-related noise a potential public health problem. Therefore, it is important to understand the magnitude of exposure and to use this information to determine associated health risks.
As a pilot study, noise levels of 10 selected locations in Toronto were measured continuously for a full week to explore temporal variability. In addition, 30-minute samples were taken over 200 locations within the Greater Toronto Area to explore the spatial variability. The objective of this study is to explore and describe the heterogeneity of environmental noise using these samples and additional information collected by Public Health Ontario.
Acknowledgements
Thanks to Ray Copes and Hong Chen, Environmental and Occupational Health, Public Health Ontario for providing this data.
Please address queries about the data to Lennon Li (Lennon.li@oahpp.ca), Analytic Services Unit, Knowledge Services, Public Health Ontario.
Background
Exposure to traffic-related noise is ubiquitous in modern society. Yet, environmental noise (originating mainly from traffic) has been implicated as a risk factor for adverse cardiovascular outcomes. A systematic review of the associations between noise from transport sources (e.g., road, air and rail) and cardiovascular diseases concluded that individuals exposed to higher levels of traffic-related noise are at increased risk for hypertension and ischemic heart disease. Acute exposure to noise is hypothesized to activate sympathetic and endocrine stress, and over a longer term it may result in permanent vascular changes such as increasing stress hormones and blood pressure, which may predispose individuals to arterial hypertension.
Thus, exposure to traffic-related noise has the potential to pose a tremendous public health burden. Indeed, in a recent report from World Health Organization it is estimated that 3% of myocardial infarction cases (or 1629 new cases) in Germany in 1999 were due to noise from road traffic. To reduce potential health impacts that may result from environmental noise in Ontario, it is important to understand the magnitude of exposure to the population and to use this information to determine associated health risks. Unfortunately, the measurement of population exposure to environmental noise is virtually nonexistent at present in Ontario.
Public Health Ontario (PHO) took its initiative and collected environmental noise data in two cycles in different seasons, see next section for detailed description. Our primary objective is to understand the spatial and temporal patterns in the data collected in Cycle 1. Our secondary objective is to see how the Cycle 2 data compares with the Cycle 1 data, refer to the objective section for details.
Data collection
Ideally, measurements taken over time and at multiple locations that cover the entire area are needed. However, this needs to be balanced with time constraints, equipment safety and human resources. Two sampling strategies were used to balance these competing needs: a) convenience sampling on weeklong sites to explore temporal variability, a total of 10 sites were selected for continuous measurement for a full week. b) Lattice (regular grids) with close pairs sampling on short-term monitoring sites to explore spatial variability. A total of 70 equally spaced sites (3km apart) were selected to ensure of the coverage of Great Toronto Area, and another 130 random locations were generated for mobile sampling. To avoid systematic bias due to sampling date, Toronto is divided into 40 regular grids with approximately 5 sites per grid and sampling order of each grid is randomized. Additional 41 locations were selected within 200 meters from these 200 sites to capture spatial correlation in short distances. As a result, a total 241 locations were sampled within Greater Toronto Area with measurement of 30 minutes each during 9am-5pm on week days. Information on traffic count and land use etc. for each site was also collected.
A sound meter was used to measure the level of noise at each sampling site. The measurements were taken continuously (fractions of a second) for 30 minutes at mobile sampling site and a week at weeklong sampling site. The meter measures average sound pressure levels and then converts those into a log-scale decibel reading (dB). Because the measurements are continuous, the data need to be average onto different time units (e.g., seconds, minutes) for output and analysis, this average is called Leq (equivalent steady sound level of a noise energy-averaged over time, see page 3 of this document). Note that this average is not the arithmetic mean of the raw measurement data therefore it is not sensible to take the arithmetic means of the Leqs. The datasets provided here give 30 minutes average Leqs for both mobile and weeklong sampling sites using the following formula:
where Li = level reading i (dB) at a fraction of a second and n = total number of readings in 30 minutes. You could directly use the Leqs in the data as the outcome in the analysis.
Principal
Pour une étude pilote, l’objectif principal est de comprendre la variabilité spéciale et temporelle du bruit dans la grande région de Toronto en utilisant des mesures prises sur des période d’une durée d’une semaine (Weekly_Sample.csv) et données mobiles collectées au cycle 1 (Cycle1_mobile.csv). Plus précisément,
- Décrire les variations hebdomadaires du niveau de bruit à Toronto; Est-ce que les torontois sont exposés à plus de bruit à certaines heures de la journée et/ou certains jours de la semaine.
- Décrire les variations spatiales; est-ce que certaines localisations de Toronto sont exposées à des niveaux plus élevés de bruits que d’autres ? Comment est-ce les caractéristiques (intensité du trafic, utilisation du territoire, …etc) contribuent aux variations observée ?
Secondaire
On a aussi collecté des données à 313 sites additionnels durant une autre saison de l’année. Parmi ceux-là, 100 sites sont ré-échantillonnés parmi ceux sélectionnés au cycle 1 (Cycle1_rep.csv) et 213 sites sont nouveaux (Cycle2_Mobile.csv).
Utiliser cette base de données pour valider, calibrer et mettre à jour vos résultats. Plus précisément
- Est-ce que les variations spatiales observées au cycle 2 sont similaires à celles observées au cycle 1?
- Si les variations diffèrent d’un cycle à un autre, explorer les raisons et sources, sinon mettre à jour les résultats.
1. WeeklySample (CSV)
- SiteID: Identité du site à long terme échantillonné.
- Time: Moment de mesure à une demie-heure.
- LEQ: Moyenne des niveaux de bruit à chaque 30 minutes.
2. Cycle1_Mobile, Cycle2_Mobile & Cycle2_rep (CSV)
- SiteID: Identité des sites mobiles.
- GridID: Grille des sites appartenant à, Grille est seulement utilisée à des fins de randomisation.
- LEQ: Moyenne des niveaux de bruit sur 30 minutes.
- Date: Date de prise de l’échantillon.
- Start.time: Heure début de l’utilisation du sonomètre.
- End.time: Heure fin de l’utilisation du sonomètre.
- Lat: Latitude du site
- Long: longitude du site
- Total.traffic: Nombre total de véhicules qui passent Durant la période d’échantillonnage.
- Distexp: Distance à la plus proche autoroute.
- Com100re: Surface totale à utilisation commerciale à l’intérieur des 100 mètres.
- Ind100re: Surface totale à utilisation industrielle à l’intérieur des 100 mètres.
- Open100re: Total de surface ouverte à 100 mètres
- Rec100re: Surface totale à utilisation récréative à l’intérieur des 100 mètres.
- Res100re: Surface totale à utilisation résidentielle à l’intérieur des 100 mètres.
- Wat100re: Surface des cours d’eau à l’intérieur des 100 mètres.
- Pop2001_500m: Densité de la population à l’intérieur des 100 mètres.
Pour accéder aux jeux de données, vous devez télécharger le présent document de confidentialité , l’imprimer, le signer et le retourner à Lennon Li (Lennon.li@oahpp.ca). Il vous enverra alors les jeux de données par courriel. Un petit échantillon de ces données se trouve ici.
- Van Kempen E, Babisch W. The quantitative relationship between road traffic noise and hypertension: a meta-analysis. J. Hypertens. 2012 Jun;30(6):1075–86.
- Sørensen M, Andersen ZJ, Nordsborg RB, Jensen SS, Lillelund KG, Beelen R, et al. Road Traffic Noise and Incident Myocardial Infarction: A Prospective Cohort Study. PLoS ONE. 2012 Jun 20;7(6):e39283.
- Gan WQ, Davies HW, Koehoorn M, Brauer M. Association of long-term exposure to community noise and traffic-related air pollution with coronary heart disease mortality. Am. J. Epidemiol. 2012 May 1;175(9):898–906.
- Babisch W. Road traffic noise and cardiovascular risk. Noise Health. 2008 Mar;10(38):27–33
- Diggle, Peter J., Ribeiro Jr, Paulo Justiniano, Model-based Geostatistic, Springer Series in Statistics, 2007
- Royster, Berger, Royster. The Noise Manual. American Industrial Hygiene Association, Akron, Ohio, 2003.
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